Salt and Blood Pressure on a Keto Diet


You go to any old school doctor and they’re going to tell you that if your blood pressure’s high, you need to decrease your sodium intake. Well, that’s living in the past. Science has changed. Everything has evolved
in the world of our diets and in the world of health in general. So let’s talk today
about your blood pressure and what’s really causing
this rise in blood pressure. I’ll tell you it’s not salt. Hey I’m Thomas DeLauer with Keto-Mojo and let’s get into some keto science in the world of blood pressure. So first off we have
to know the importance of lower blood pressure. There are a lot of links that show that if your blood pressure is lower, you’re going to live longer. There was a study that was published in the New England Journal of Medicine. Okay this was a big study. It took a look at 10,000 participants over a number of years and overall they found that in general, if subjects had a blood
pressure of 120 or lower with their systolic blood pressure, they on average lived 21% longer. So this doesn’t necessarily mean that having low blood pressure
is the end all be all. There could be a lot of
other causing factors there that caused the blood pressure
to go up in the first place. But, we do know that generally speaking, if your blood pressure is lower, you’re usually a healthier person. Now to understand blood
pressure really quick we have to look at systolic and diastolic. Systolic is the blood pressure when your heart is contracting. So when your heart beats, it sends blood throughout your body. This systolic blood pressure is the pressure of the blood
pushing out on the arteries right after the heart beats, okay. Then we have diastolic blood pressure. That’s sort of the opposite. It’s when your heart is not beating. It’s when it’s relaxing and it’s the blood
pressure that is exerted on the artery at that point in time. So that’s how we know
kind of the difference between the two. Now let’s talk about salt for a second. Salt is not necessarily the culprit. You see, salt does play a
big role in fluid retention, so yes, if you do consume some salt, you might see an increase
in your blood pressure. But it is not the end all be all. In fact, there are a lot
of other indirect things that sort of act as a
catalyst to make salt have a more profound effect. You see the bigger problem
comes down to carbohydrates and comes down to sugar in general and really just insulin. You see, there’s something
known as Hyperinsulinemia. Hyperinsulinemia is
when your insulin levels are chronically elevated. So someone that consumes a lot
of carbohydrates all the time is going to have a high level of insulin. This high level of insulin
does something unique to the kidneys. You see, it tells the kidneys to hold on to water and sodium. Normally one of the jobs of the kidneys is to expel sodium and water but if we have lots of insulin, the kidneys sort of do the opposite. They tell the body to hold onto it. Well, when the kidneys
are telling the body to hold on to water and sodium, of course your blood
pressure’s going to increase. You’re going to have more
water volume in your blood so you’re going to have more blood. You’re going to have more sodium so you’re going to have an expansion of that. So of course your blood
pressure’s going to go up. Now, if you were to have
lower levels of insulin, your kidneys would be excreting the water and the salt and your blood pressure would go down. You see there’s another factor and this is called insulin resistance. If you eat a lot of
carbs or a lot of sugar for an extended period of time, you start to become what is
called insulin resistant. Insulin resistant is just
like the name implies. It’s where your body doesn’t really register insulin anymore. It becomes sort of blunted to insulin. So what ends up happening there is the cells don’t absorb
things nearly as well. That’s why diabetic people end up with higher blood sugar. They’re not absorbing sugar. But, little do people know, it makes it so that you
don’t absorb magnesium either and magnesium plays a very critical role in the muscles being able
to relax within the body and that includes muscles that
are in our arterial walls. So it allows muscles
and arteries to relax. A relaxed muscle and a relaxed artery ends up having a lower blood pressure because it’s not all tight
and tense all the time. Now another thing that this does when you’re lacking magnesium or you’re not absorbing other minerals, is it disrupts cellular communication. If you don’t have proper
cellular communication or a proper electric system
as I like to call it, you’re never going to be able
to get the right signaling to increase or decrease
blood pressure properly. Now there’s other things
we have to factor in too. Like when our magnesium
levels are all out of whack, so are our sodium and
our potassium levels. So without absorbing magnesium, we’re not able to have the right effect. Our minerals just
completely get out of whack and therefore our blood pressure all gets totally cattywampus! And there’s one other
piece to the equation and that’s the fructose
nitric oxide corelation. You see with fructose, or more commonly high fructose corn syrup, the body has to metabolize
at 100% in the liver. Now this metabolism in the
liver causes a disruption in the production of nitric oxide. Nitric oxide is what increases blood flow. If blood can move freely, blood pressure can be controlled. So, if fructose slows down the
production of nitric oxide, then we have restricted blood flow, which means the blood
volume is still there but it’s restricted and this vasoconstriction means that the blood pressure elevates. So now we look at all this. How do we connect the dots with keto? Well, there’s one study that was published in the Archives of Internal Medicine and this study really puts it all together and makes it very, very, very obvious that the keto diet, or a least a lower carb diet, could be the potential solution
to high blood pressure. So this study took a look at 146 subjects, broken into 2 groups. One group did a keto diet with less than 20 grams of carbs per day and another group ate a more
conventional, low calorie diet, along with prescription
weight loss medications. Both groups on average had a
500 to 1000 calorie deficit. Okay, so they were both
in a weight loss category where they’re trying to lose weight. But what researchers wanted to look at was which one had a better
effect on blood pressure. Well, when it came down to
the systolic blood pressure, they found that the keto diet on average had a decrease of 5.9
millimeters per mercury (mmHg) versus 1.5 mmHg in the non-keto group. So 5.9 mmHg versus 1.5 mmHg. Huge difference right then and there. Now when it came down to
the diastolic readings, the keto group saw a decrease
in 4.5 mmHg where the non-keto group
only saw a decrease of 0.4 mmHg. So, that’s pretty obvious
right there, right. And a lot of it has to do with
what I already stated before. The insulin, the overall carb intake, and the overall retention
of sodium and water. So when you consume extra
salt on a keto diet, it doesn’t really effect
your blood pressure because you’re not having
the carbs as the catalyst to tell your body to hold onto it. But how do you know if
you’re in a keto diet? How do you know if you’re
really doing it right? Well again, that is exactly where utilizing the
Keto-Mojo meter comes in. You don’t want to just be
guessing all the time. You don’t want to just say uh
I’m reducing my carb intake. I am on the road to being healthier. I mean that’s a good start but when it comes down
to getting granular, you want to be measuring. And the Keto-Mojo meter will tell you exactly where
your blood levels are at of ketones, specifically beta hydroxybutyrate, which is the main ketone body. So you have to rely on
data and rely on science. You can’t just float through life using guess work all the time. So make sure you check
out the Keto-Mojo meter, leave the guesswork out of the equation and use the actual measuring to the meter. So as always, keep it locked in here with Keto-Mojo and I’ll see you in the next video.

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